GI Stability™

^To date, shown in multiple animal studies, infants, and one adult human study.

Digestive health issues affect daily life for millions of Americans. Many of these conditions are paired with a disrupted microbiome,¹ which throws the GI system out of balance and may cause gut distress. Individual lifestyles, diets, antibiotic use, and traveling patterns have the potential to disrupt the microbiome. Building resilience to disruption is important – it helps give the human microbiome the ability to return to equilibrium.² GI Stability provides targeted support for protecting the complex gastrointestinal environment.

How GI Stability Works

GI Stability takes a systems-based approach to managing the microbiome and supporting a healthy GI environment. It contains 2’-FL – a unique prebiotic HMO (Human Milk Oligosaccharide) that resists digestion and moves directly to the lower GI tract where it becomes effective in feeding selective bacteria.³

Studies show that HMOs have a unique structure that is preferred by beneficial microbes who use them to grow.^4,5

The Importance of HMOs

HMOs are naturally found in human milk and contribute to the benefits of breast-feeding that are linked to long term development, immune protection, and microbial population cultivation.^5,6 These benefits are not exclusive to infants, though. GI Stability is a great source for targeted prebiotic action that contributes to a healthy microbiome and may provide immune system support.*

GI System Health and Immune System Support

Proper GI integrity and a healthy gut microbiome help support immune defenses by influencing the development and production of immune cells, and by helping to regulate local GI and systemic immune system responses.^7,8 Sustaining these microbes and supporting the cells that make up the GI lining with selective prebiotics is essential for GI support in the face of acute and chronic GI stress.^7,8

Citations: 

1. Chan, Y.K., et al. Annals of Nutrition and Metabolism, 2013. 63(suppl 2)(Suppl. 2): p. 28-40.
2. Ruan, W., et al. Digestive Diseases and Sciences, 2020: p. 1-11.
3. Gibson, G.R., et al. Nat Rev Gastroenterol Hepatol, 2017. 14(8): p. 491-502.
4. Yu, Z.-T., C. Chen, and D.S. Newburg. Glycobiology, 2013. 23(11): p. 1281-1292.
5. Bode, L. Glycobiology, 2012. 22(9): p. 1147-1162.
6. Marcobal, A., et al. J Agric Food Chem, 2010. 58(9): p. 5334-40.
7. Shukla, S.D., et al. Clinical & Translational Immunology, 2017. 6(3): p. e133.
8. Budden, K.F., et al. Nature Reviews Microbiology, 2017. 15(1): p. 55-63.